Identifying and Recognizing Lyme Disease
Copyright (c) 2010 John Howe
Lyme Disease in 2010 is no longer a guessing game.
1975 was the first recognition of the condition in Lyme, CT. For those afflicted with the symptoms, the problem of bringing Lyme disease to resolution has been to accurately identify the disease. However, the history of the disease goes back to 1883 as you can see by the graph below provided by Brown University relying on information provided in part by Douglas Dodge (See references below). Since the formal recognition and naming of the disease, Lyme has spread and continues to do so at an alarming rate.
Timeline of important events in the history of Lyme Disease
1883 – The first recorded case of Lyme Disease is described by Alfred Buchwald. He described it as a degenerative skin disorder now known as ACA (acrodermatitis chronica atorphicans).
1909 – Arvid Afzelius presents his research on expanding ring like lesion (erythema migrans) associated with what would become known as Lyme Disease.
1921 – Arvid Afzelius publishes his work and speculates on the origin of the disease as the Ixodes Scapularis tick and connects the disease with joint problems.
1922 – The disease is found to be associated with neurological problems.
1930 – Psychiatric problems are found to also be caused by the disease.
1934 – Appearance of EM or ACA is associated with benign lymphocytomas. Arthritic symptoms are reported in connection with the disease.
1970 – Rudolph Scrimenti, professor of dermatology publishes reports of the rash.
1975 – Allen C. Steere, Yale, investigates a group of rashes and swollen joints in Lyme, Connecticut. Scrimenti publishes about his treatment of the rash with penicillin.
1976 – A clustering of cases of the disease is reported in a Naval Medical Hospital in Connecticut.
1977 – Steere et. Al misdiagnoses Lyme Disease as juvenile rheumatoid arthritis and names the condition Lyme Arthritis.
1982 – The etiological agent of the disease, Borrelia burgdorferi, is discovered by Dr. Willy Burgdorfer.
1983 – Treatment of Lyme disease with oral and intravenous antibiotics begins and clinicians define the three stages of Lyme Disease.
1986 – Skeptics denounce the existence of Lyme Disease.
1988 – Lyme Disease Foundation (LDF) Founded.
1997 – Borrelia burgdorferi genome sequenced by TIGR.
1999 – Smith Kline Beecham receive FDA approval and place LYMErix vaccine on the market.
2002 – LYMErix vaccine removed from market.
Blood test provided the first means of identifying Lyme Disease. Unfortunately, a large percentage of the results generated from this method were found to be inaccurate.
2010 – Of most recent importance is the following…
According to an article published March 30th, 2010 entitled Connecticut Pathologist Debuts Lyme Disease Test Based on Nested PCR, DNA Sequencing, by Kirell Lakhman, “A scientist in Connecticut has launched a test that uses nested PCR and DNA sequencing to detect the presence of Borrelia burgdorferi, the bacterium that causes Lyme disease.
Prior to the onset of the tick season of 2010, the test, referred to as LoTemp, is being made available. Other PCR-based tests for the indication exist, “{b}ut this is the first … one using nested PCR for detection and DNA sequencing to validate the molecular diagnosis in clinical laboratory medicine.”
The assay, developed by Sin Hang Lee, a pathologist at Milford Hospital in Milford, Conn., can be used before patients undergo traditional serology testing for the bug. In the Unied States, it is suspected that more than 30,0000 are infected with spirochete B. burgdorferi each year.
The test uses nested PCR to detect genomic DNA of B. burgdorferi in blood, and uses Sanger-based DNA sequencing and diagnostic signature sequences found in GenBank to validate the result, according to the statement. (The polymerase chain reaction (PCR) is a scientific technique in molecular biology to amplify a single or a few copies of a piece of DNA across several orders of magnitude, generating thousands to millions of copies of a particular DNA sequence.)
Up to 75 percent of patients with “acute-phase Lyme disease are negative for the characteristic antibodies, but in fact the percentage is higher,” the statement said. However, a negative result does not rule out the presence of the bacteria because spirochetlemia, or the presence of spirochetes in the blood, “is transient and its time points in Lyme {disease} vary from patient to patient.”
“It is the marriage of {nested PCR and DNA sequencing} that minimizes false-negatives to the lowest possible and eliminates false-positives known to be associated with other Lyme disease DNA tests,” it said.”
The point is that the approach to Lyme disease detection and resolution has relied on traditional allopathic approaches. However, Lyme Disease has a distinct energetic signature and a process we call Bio-communications Diagnostics is designed to detect and measure that energetic signature response. During a Bio-Communications Diagnostic session, a computer generated stressor signal is send to the body. In response to the stressor signal, an energetic signature signal is received back. This response provides an energetic measurement showing the presence of Lyme related bacterium associated with the Lyme Spirochetes. From this response, the presence of Lyme can be confirmed and the Lyme bacterial load level in the body can be determined.
2010, the good news is that the presence of Lyme in the body no longer has to be a guessing game, nor does resolution of the disease have to include antibiotics.
References:
Brown University Lyme Disease History – http://www.brown.edu/Courses/Bio_160/Projects2005/lyme_disease/references.htm
For more than 20 years, John Howe has been studying the relationship of energy in human beings to personal health. For more information see Lyme Disease Identification and Resolution and PeterHowe-Healer.com.
Article from articlesbase.com
Hyperbaric Chambers & Lymes Disease
Lymes Disease
“Lyme disease is a bacterial infection caused by a spirochete that is transmitted to humans by a tick bite. Lymes Disease usually followed by a bulls eye rash and erythema migrans, with many other flu like symptoms such as painful joints and muscles, headaches, fatigue, swollen glands, sore throat, fever, mental fog and many more. If left untreated, Lymes Disease can become a severely debilitating disease affecting both neurological and motor abilities. Lymes Disease is recommended that antibiotic therapy be used in conjunction with hyperbaric oxygen and has shone great response, allowing many patients to live more productive lives. Recommended treatment is a 60 minute treatment twice a day, five days a week followed by a two day rest. Lymes Disease is recommended a series of 30-60 treatments. Patients develop a Jarisch-Herxheimer reaction usually within the first week of treatment which is the Lyme bacteria die-off.
The United States and parts of Europe have seen rising numbers of Lyme disease infections because it was found thirty years ago. There’s one particular class of micro organism known as a spirochete that’s liable for the development of this Lymes Disease. Lyme disease may sound like an odd title, but it surely was truly named after the town in Connecticut where it was initially discovered. At first Lymes Disease was mistaken for rheumatoid arthritis, but Lymes Disease surely was determined that there had to be something else when a gaggle of youngsters from totally different properties have been all coming down with it on the similar time. It was formally coined, “Lyme Illness,” in 1982 after the bacterial cause was determined.
Lymes Disease
is the chew of an infected tick that causes you to develop Lyme. It is this bite that permits the micro organism to enter your body. Lymes Disease isn’t a contagious illness the way that the flu is, as that chew is necessary for infection.
Every individual is going to develop Lymes Disease symptoms in a different way. The spherical “bull’s eye” rash is a symptom of Lymes Disease that could be very recognizable. Not everybody with Lymes Disease will develop this rash, however those that do normally start seeing it within a number of days. Lymes Disease can develop for a few weeks before it fades away by itself. Lymes Disease is very important notice that despite the fact that the rash will fade by itself, the illness is not going to; Lymes Disease requires medical treatment with the intention to remedy properly. Other symptoms of Lymes Disease that you may discover are malaise, joint pain, and extra rashes.
Following the chew, it may be days or even weeks before you begin exhibiting any Lymes Disease symptoms. The Lymes Disease symptoms of lupus and fibromyalgia are very similar to these of Lyme, so should you do not know that a tick chew has occurred, you might be misdiagnosed as having one of many other conditions. A blood test is usually necessary as a way to ensure that Lymes Disease is present.
Lymes Disease
requires therapy as a result of allowing it to worsen over time will cause some very serious problems. If it isn’t treated,
Lymes Disease can become fairly debilitating in the later stages
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Morgellons Disease & Lymes Disease
Morgellons Disease & Lymes Disease
It’s hard to say yet which pattern the Morgellons phenomenon will follow. Will it be the next Lyme disease, validated by the medical community? Or will its victims reside in diagnostic purgatory forevermore? Medicine is full of phenomena that sounded like psychological ailments when first proposed but are now linked to invasive pathogens.
Ulcers are caused by spirochetes, not stress. Syphilis is a bacterial infection that reaches the brain, not a kind of insanity. Tuberculosis is another infectious disease, not the psychosomatic illness it was first thought to be.
But while these stories become bits of medical lore, and the names of the scientists who made the discoveries are now well-known, the stories of the patients who were caught in the middle of the confusion are rarely told. Mary Leitao says she never wanted to be an activist. “I’m just a scared mother,” she says. And she wants an answer, whatever it is, soon.
Not long after Drew became symptomatic, his two older siblings, Jeremy, 16, and Samantha, 17, started exhibiting signs of it, too. Samantha is plagued by cognitive problems, fatigue, and joint pain. She was once in the gifted program; but now, in her senior year of high school, just started homebound instruction. Jeremy also suffers from fatigue and some cognitive problems. But he pushes himself to get to school. “I’m not sure how much longer he can push himself,” says Leitao. Drew, now 8, doesn’t seem to be cognitively afflicted, but his skin, she says, is riddled with sores and scars.
There are other families in which more than one person suffers from the symptoms too, says Leitao. But rather than see that as an indication of the disease’s infectious nature, the medical community has labeled it “folie a famille” (madness of the family). They have, she says, an explanation for everything.
